Can hypothyroidism cause acute central serous chorioretinopathy?

dc.authorid0000-0003-4468-3985en_US
dc.authorid0000-0003-2544-3580en_US
dc.authorid0000-0002-4542-4064
dc.authorid0000-0001-8305-6600
dc.contributor.authorUlaş, Fatih
dc.contributor.authorUyar, Enes
dc.contributor.authorTekçe, Hikmet
dc.contributor.authorÇelebi, Serdal
dc.date.accessioned2021-06-23T19:50:41Z
dc.date.available2021-06-23T19:50:41Z
dc.date.issued2019
dc.departmentBAİBÜ, Tıp Fakültesi, Cerrahi Tıp Bilimleri Bölümüen_US
dc.description.abstractPurpose: To evaluate selected systemic findings, especially thyroid functions, in acute central serous chorioretinopathy (CSC) patients. Materials and Methods: In all, 71 consecutive acute CSC patients who fulfilled the inclusion criteria and 70 age-matched healthy control subjects were included in the study. Systemic findings, including serum levels of thyroid hormones, thyroid stimulating hormone (TSH), mean arterial pressure (MAP), pulse rate, serum lipid levels and optical coherence tomography findings, were compared between the groups. Independent samples t-test was used for statistical analysis. Results: The mean ages of the CSC and control groups were 41.06 +/- 6.49 and 40.06 +/- 7.08 years old, respectively. Retinal thickness, choroidal thickness, TSH levels, pulse rate and MAP were significantly different between CSC patients and healthy control subjects (range of p values: <0.001-0.042). In the logistic regression analysis, MAP, serum triglyceride concentration and central choroidal thickness were positively associated with CSC (range of p values: <0.001-0.035). Conclusion: Acute CSC patients had significantly higher pulse rates and MAP and significantly thicker choroidal thickness than were found in healthy subjects. TSH levels were also significantly higher in CSC patients than in controls. Hence, hypothyroidism might be associated with CSC.en_US
dc.identifier.doi10.1080/08820538.2019.1684524
dc.identifier.endpage540en_US
dc.identifier.issn0882-0538
dc.identifier.issn1744-5205
dc.identifier.issue7-8en_US
dc.identifier.pmid31646925en_US
dc.identifier.scopus2-s2.0-85074587084en_US
dc.identifier.scopusqualityQ2en_US
dc.identifier.startpage533en_US
dc.identifier.urihttps://doi.org/10.1080/08820538.2019.1684524
dc.identifier.urihttps://hdl.handle.net/20.500.12491/9838
dc.identifier.volume34en_US
dc.identifier.wosWOS:000492108000001en_US
dc.identifier.wosqualityQ4en_US
dc.indekslendigikaynakWeb of Scienceen_US
dc.indekslendigikaynakScopusen_US
dc.indekslendigikaynakPubMeden_US
dc.institutionauthorUlaş, Fatih
dc.institutionauthorUyar, Enes
dc.institutionauthorTekçe, Hikmet
dc.institutionauthorÇelebi, Serdal
dc.language.isoenen_US
dc.publisherTaylor & Francis Incen_US
dc.relation.ispartofSeminars In Ophthalmologyen_US
dc.relation.publicationcategoryMakale - Uluslararası Hakemli Dergi - Kurum Öğretim Elemanıen_US
dc.rightsinfo:eu-repo/semantics/closedAccessen_US
dc.subjectCentral Serous Chorioretinopathyen_US
dc.subjectChoroiden_US
dc.subjectHypertensionen_US
dc.subjectHypothyroidismen_US
dc.subjectMean Arterial Pressureen_US
dc.subjectThyroid Stimulating Hormoneen_US
dc.titleCan hypothyroidism cause acute central serous chorioretinopathy?en_US
dc.typeArticleen_US

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