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    Loss of eyebrows (madarosis) after use of long-acting methylphenidate: case report
    (Lippincott Williams & Wilkins, 2017) Yektaş, Çiğdem; Samurcu, Nuran Demir; Tufan, Ali Evren
    To the Editors: “Madarosis” from the Greek root of “madao” (shedding) is a term in medical terminology used to define loss of eyebrows and/or eyelashes due to shedding. Madarosis can arise as a clinical sign of diverse local or systemic disorders with various etiologies. The medical causes of madarosis include infections of eye and related structures (eg, blepharitis), dermatological disorders (eg, telogen effluvium, dermatitis, psoriasis), systemic and endocrine disorders (eg, systemic lupus erythematosus, hyperthyroidism/hypothyroidism, hyperparathyroidism/hypoparathyroidism), nutritional deficiencies leading to protein and trace element deficits (eg, zinc, biotine, ferritin), infections (eg, lepra, syphilis, fungi, and various parasites), trichotillomania, local radiotherapy, chemotherapy, and some of the pharmaceuticals.1 Madarosis can be induced by diverse groups of pharmaceuticals, among which retinoids, heparine, antiepileptics, inhibitors of the angiotensin-converting enzyme, androgenes, miotics, anticoagulants, lithium, propranolol, bromocriptine, statins, and antithyroid agents are listed. Those may lead to shedding and loss of the eyelashes and eyebrows in the telogen phase.
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    Response of catatonia to amisulpride and lorazepam in an adolescent with schizophenia
    (Mary Ann Liebert, Inc, 2018) Binici, Nagihan Cevher; Topal, Zehra; Samurcu, Nuran Demir; Cansız, Mehmet Akif; Savcı, Ugur; Öztürk, Yusuf; Özyurt, Gonca
    Letters to the editor
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    Social communication disorder: A narrative review on current insights
    (Dove Medical Press Ltd, 2018) Topal, Zehra; Samurcu, Nuran Demir; Taşkıran, Sarper; Tufan, Ali Evren; Semerci, Bengi
    Social communication disorder (SCD) is a novel diagnosis listed under the rubric of communication disorders within the Diagnostic and Statistical Manual of Mental Disorders-5 (DSM-5) and it is reported to be characterized by impairment in use of verbal and nonverbal communication for social aims. This review attempts to summarize the current understanding of the SCD concept along with its evolution and presents data from previous studies conducted. Suggestions for further research are also delineated. As listed in DSM-5, the criteria for this novel diagnosis are vague, display elevated comorbidity with other neurodevelopmental disorders and other childhood psychopathologies, and show partial overlap with autistic spectrum disorders both in terms of genetics and family histories. Data on cross-cultural presentations and temporal stability are also limited. The social communication model proposed by Catani and Bambini may help integrate the neurobiological findings pertaining to SCD. Valid and reliable assessment methods need to be developed for SCD. This may involve either development of novel instruments capturing the DSM-5 criteria or application of statistical methods such as item response theory to existing instruments. The relationships between broad autism phenotype, pragmatic language impairment, nonverbal learning disorder, learning disorders, autistic spectrum disorders, and SCD should be evaluated with further studies.